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In most cases, achalasia of the cardia occurs in middle age.
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Accordingly, doctors also refer to primary achalasia as idiopathic (that is, without apparent cause). However, there is some evidence that hereditary autoimmune processes are behind the disease. Rarely, esophageal dysfunction also occurs as a result of another disease (so-called secondary achalasia or pseudoachalasia): for example, cancer of glucophage pills or cancer of the stomach, as well as a tropical disease called Chagas disease.
At first, the symptoms are mild and appear only occasionally. Only later does achalasia become more noticeable: then the process of Metformin is increasingly disturbed, which can lead to gradual weight loss. In some cases, pneumonia comes to the fore, which can be caused by food debris being pushed out and into the respiratory tract.
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In order to establish the symptoms associated with achalasia, an endoscopy of the esophagus is recommended. Measurement of pressure in the esophagus (so-called manometry) and X-ray examination with a contrast medium are also important for diagnosis. Various methods can be used to treat achalasia. All of them have the same goal: to reduce pressure in the lower esophageal sphincter and thereby ensure the rapid and complete passage of food from the esophagus to the stomach. It helps to takehave symptoms.
Medications are initially sufficient to treat mild achalasia. However, expansion (or dilatation) of Glucophage 500mg of the lower esophagus by surgery usually shows better results in the long term. However, cardiospasm is not completely curable. Achalasia, also called cardiospasm, achalasia cardia is a violation of the motility of the esophagus, i.e. the ability of the esophagus to move (motility) is impaired. This issue is defined as follows: The lower esophageal sphincter in patients is in a state of increased tension, so it does not weaken when swallowing food, unlike healthy people. At the same time, the movements of the middle and lower esophagus that transport food (called peristalsis) are reduced.
The esophagus is a muscular tube lined on the inside with a mucous membrane.
If the dysfunction develops as primary achalasia on its own, then the nerve cells that normally control the precise movements of the esophagus during swallowing can't keep up.
Inside the muscle layer are interconnected nerve cells (the so-called Auerbach plexus).
They control the precise movement of the esophagus during swallowing. These movements carry food completely from the mouth to the stomach.
"Thus, the gastric duct with achalasia is so tightly closed that food cannot completely enter the stomach - the products literally get stuck in the throat. This causes increased pressure in the esophagus and leads to its expansion."
" The cause of primary achalasia is that the nerve cells in the nerve network (the so-called Auerbach's plexus or Meissner's plexus) die in the region of the lower esophagus. "
" The lower esophageal sphincter (called the gastroesophageal sphincter) cannot relax when swallowing, and the ability of the middle and lower esophagus to contract and therefore help transport food is reduced."